Search results for "Aurora B kinase"

showing 2 items of 2 documents

Abnormal mitotic spindle assembly and cytokinesis induced by D-Limonene in cultured mammalian cells

2013

D-Limonene is found widely in citrus and many other plant species; it is a major constituent of many essential oils and is used as a solvent for commercial purposes. With the discovery of its chemotherapeutic properties against cancer, it is important to investigate the biological effects of the exposure to D-Limonene and elucidate its, as yet unknown, mechanism of action. We reported here that D-Limonene is toxic in V79 Chinese hamster cells in a dose-dependent manner. Moreover, to determine the cellular target of D-Limonene, we performed morphological observations and immunocytochemical analysis and we showed that this drug has a direct effect on dividing cells preventing assembly of mito…

Genome instabilityCell SurvivalHealth Toxicology and MutagenesisAurora B kinaseAntineoplastic AgentsSpindle ApparatusBiologyToxicologySeptinMicrotubulesGenomic InstabilityCell LineChromosome segregationInhibitory Concentration 50MicrotubuleChromosome SegregationCricetinaeCyclohexenesGeneticsAnimalsMitosisGenetics (clinical)genomic instability damage-induced mutagenesis mitosis V79 d- LimoneneCytokinesisCell DeathTerpenesAneuploidyTubulin ModulatorsSpindle apparatusCell biologySettore BIO/18 - GeneticaDrug Screening Assays AntitumorLimoneneCytokinesis
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Targeting the chromosomal passenger complex subunit INCENP induces polyploidization, apoptosis and senescence in neuroblastoma

2019

Abstract Chromosomal passenger complex (CPC) has been demonstrated to be a potential target of cancer therapy by inhibiting Aurora B or survivin in different types of cancer including neuroblastoma. However, chemical inhibition of either Aurora B or survivin does not target CPC specifically due to off-target effects or CPC-independent activities of these two components. In a previous chromatin-focused siRNA screen, we found that neuroblastoma cells were particularly vulnerable to loss of INCENP, a gene encoding a key scaffolding component of the CPC. In this study, INCENP was highly expressed by neuroblastoma cells, and its expression decreased following retinoic acid–induced neuroblastoma …

0301 basic medicineCancer ResearchINCENP/CPC/Polyploidy/DNA damage/Apoptosis/SenescenceCarcinogenesisChromosomal Proteins Non-HistoneAurora B kinaseApoptosisBiologymedicine.disease_causeArticlePolyploidy03 medical and health sciencesMiceNeuroblastoma0302 clinical medicineNeuroblastomaSurvivinmedicineGene silencingAnimalsHumansneoplasmsCellular SenescenceINCENPmedicine.disease030104 developmental biologyOncologyApoptosisTumor progression030220 oncology & carcinogenesisCancer researchHeterograftsCarcinogenesis
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